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Influences On Normal Physical Essay Research Paper

Influences On Normal Physical Essay, Research Paper


Physical growth in early childhood is partially easy to measure and


gives an idea of how children normally develop during this period. The


average child in North America is less than three feet tall at two years of


age. Physical growth contains no discrete stages, plateaus, or qualitative


changes. Large differences may develop between individual children and


among groups of children. Sometimes these differences affect the


psychological development of young children. These differences create a


nice variety among children.


Most dimensions of growth are influenced by the child’s genetic


background. Also, races and ethnic backgrounds around the world differ


in growth patterns. Nutrition can affect growth, but it does not override


genetic factors.


One factor in the cause of slow growth is malnutrition. Malnutrition


can start as early as pregnancy. Low birth weight babies have an


increased risk of infection and death during the first few weeks of life.


Food-deprived children carry a greater risk of neurological deficiencies


that result in poor vision, impaired educational attainment, and cerebral


problems. Such children are also more prone to diseases such as malaria,


respiratory tract infections or pneumonia. The illnesses of malnourished


children can cause more lasting damage than in a healthy child. The


destructive conjunction between low food intake and disease is magnified


at the level of the hungry child. There is evidence, according to The


Journal of Nutrition, that an estimated 50 percent of disease-related


mortality among infants could be avoided if infant malnutrition were


eradicated. It has also been shown that low birth- weight is associated


with increased prevalence of diseases such as stroke, heart disease and


diabetes in adult life. Most damage during the first few years of life


cannot easily be undone.


There are many reasons why some children never reach normal


height. Some causes of short stature are well understood and can be


corrected, but most are subjects of ongoing research. Achondroplasia is


the most common growth defect in which abnormal body proportions are


present. Achondroplasia is a genetic disorder of bone growth. It affects


about one in every 26,000 births. It occurs in all races and in both


sexes. It is one of the oldest recorded birth defects found as far back as


Egyptian art. A child with achondroplasia has a relatively normal torso


but short arms and legs. People sometimes think the child is mentally


retarded because they are slow to sit, stand, and walk alone. In most


cases, however, a child with achondroplasia has normal intelligence.


Children with achondroplasia occasionally die suddenly in infancy or early


childhood. These deaths usually occur during sleep and are thought to


result from compression of the upper end of the spinal cord, which can


interfere with breathing. This disease is caused by an abnormal gene.


The discovery of the gene allowed the development of highly accurate


prenatal tests that can diagnose or rule out achondroplasia. There is


currently no way to normalize skeletal development of children with


achondroplasia, so there is no cure. Growth hormone treatments, which


increase height in some forms of short stature, do not substantially


increase the height of children with achondroplasia. There is no way to


prevent the majority of cases of achondroplasia, since these births result


from totally unexpected gene mutations in unaffected parents.


One treatment available for children is known as growth hormone


therapy. The policy governing the use of growth hormone (GH) therapy


has shifted from treating only those children with classic growth hormone


deficiency to treating short children to improve their psycho social


functioning. This has caused quite a controversy. Parents have described


shorter boys as less socially competent and having more behavioral


problems than that of the normal sample. Shorter boys describe


themselves as less socially active but not having more behavioral

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problems than that of the normal group. This is according to a study


conducted by the Children’s Hospital of Buffalo and the State University


of New York at Buffalo. The researchers conclude growth hormone


therapy should not be administered routinely to all short children for the


purpose of improving their psychological health. They urge that


physicians consider both a child’s short stature and psycho social


functioning before making a referral for growth hormone therapy.


Another factor in the growth of children is their change of appetite.


Young preschoolers may eat less than they did as a toddler. This is also


when they will become more selective and choosy with the foods they


eat. These changes are normal and result from the slowing down of


growth after infancy. Preschool children simply do not need as many


calories as they did after birth. Children’s food preferences are influenced


by the adult models around them. Preschoolers tend to like the same


foods as their parents and other important adults in their lives.


Variations in growth can result from cultural and psychological


factors. Failure to thrive is defined in the class textbook as a condition in


which an infant seems seriously delayed in physical growth and is


noticeably apathetic in behavior. This condition may result from situations


that interfere with normal positive relationships between parent and child,


especially during infancy or the early preschool period. The result is a


deprived relationship that may lead the child to eat poorly or be plagued


by constant anxiety. The nervousness can interfere with sleep or the


production of growth hormones. If failure to thrive has not persisted for


too long, it usually can be reversed in the short run through special


nutritional and medical intervention to help the child regain strength and


begin growing normally again.


There are many factors that can result in slow growth in children.


Between the ages of two and five, growth slows down and children take


on more adult bodily proportions. Usually growth is rather smooth during


the preschool period. Genetic and ethnic backgrounds affect its overall


rate, as do the quality of nutrition and children’s experiences with illness.


Children’s appetites are often smaller in the preschool years than in


infancy, and preschoolers become more selective about their food


preferences. If children fall behind in growth because of poor nutrition or


hormonal deficiencies, they often can achieve catch-up growth if slow


growth has not been too severe or prolonged. A few children suffer from


failure to thrive, a condition marked by reduced physical growth, possibly


as a result of family stress and conflict. Bibliography


Achondroplasia. Public Health Education Information Sheet.


Http://www.noah.cuny.edu/pregnancy/march_of_dimes/birth_de


fects/achondro.html.


Byers, T. 1995. The Emergence of Chronic Diseases in Developing


Countries. SCN News 13: 14-19; Golden, M. H. N. 1995. Specific


deficiencies versus growth failure. SCN News 12:10-14.


Growth Hormone: Not for All Short Children. Medical Sciences


Bulletin, Pharmaceutical Information Associates, Ltd.


Http://www.pharmingo.com/pubs/msb/grhorm.html.


Mason, J. B. 1990. Malnutrition and Infection. SCN News. 5:


2o21; UN Administrative Committee on Coordination-Sub Committee


on Nutrition (ACC/SCN). 1995. Maternal Nutrition and health: A


Summary of Research on Birth weight. Maternal Nutrition and Health 14


(1/2): 14-17.


Pelletier, D. 1995. The Effects of Malnutrition on Child


Mortality in Developing Countries. Bulletin of the World Heath


Organization 73 (4); Pelletier, D. 1994. The Relationship between Child


Anthropometry and Mortality in Developing Countries. The Journal of


Nutrition. Supplement 124 (1OS).


Pollitt, E. 1995. Nutrition in Early Life and the Fulfilment of


Intellectual Potential. The Journal of Nutrition. Supplement 125 (4S):


1111S- 1118S.


Seifert, Kelvin L. and Robert J. Hoffnung. Child and Adolescent


Development. 1997, Chapter 8, pages 236-244.

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