РефератыИностранный языкThThe Heart Essay Research Paper CONTENTS3

The Heart Essay Research Paper CONTENTS3

The Heart Essay, Research Paper


CONTENTS


3 Introduction


4 The Human Heart


5 Symptoms of Coronary Heart Disease


5 Heart Attack


5 Sudden Death


5 Angina


6 Angina Pectoris


6 Signs and Symptoms


7 Different Forms of Angina


8 Causes of Angina


9 Atherosclerosis


9 Plaque


10 Lipoproteins


10 Lipoproteins and Atheroma


11 Risk Factors


11 Family History


11 Diabetes


11 Hypertension


11 Cholesterol


12 Smoking


12 Multiple Risk Factors


13 Diagnosis


14 Drug Treatment


14 Nitrates


14 Beta-blockers


15 Calcium antagonists


15 Other Medications


16 Surgery


16 Coronary Bypass Surgery


17 Angioplasty


18 Self-Help


20 Type-A Behaviour Pattern


21 Cardiac Rehab Program


22 Conclusion


23 Diagrams and Charts


26 Bibliography


INTRODUCTION


In today’s society, people are gaining medical knowledge at


quite a fast pace. Treatments, cures, and vaccines for various


diseases and disorders are being developed constantly, and yet,


coronary heart disease remains the number one killer in the


world.


The media today concentrates intensely on drug and alcohol


abuse, homicides, AIDS and so on. What a lot of people are not


realizing is that coronary heart disease actually accounts for


about 80% of all sudden deaths. In fact, the number of deaths


from heart disease approximately equals to the number of deaths


from cancer, accidents, chronic lung disease, pneumonia and


influenza, and others, COMBINED.


One of the symptoms of coronary heart disease is angina


pectoris. Unfortunately, a lot of people do not take it


seriously, and thus not realizing that it may lead to other


complications, and even death.


THE HUMAN HEART


In order to understand angina, one must know about our own


heart. The human heart is a powerful muscle in the body which is


worked the hardest. A double pump system, the heart consists of


two pumps side by side, which pump blood to all parts of the


body. Its steady beating maintains the flow of blood through the


body day and night, year after year, non-stop from birth until


death.


The heart is a hollow, muscular organ slightly bigger than a


person’s clenched fist. It is located in the centre of the chest,


under the breastbone above the sternum, but it is slanted


slightly to the left, giving people the impression that their


heart is on the left side of their chest.


The heart is divided into two halves, which are further


divided into four chambers: the left atrium and ventricle, and


the right atrium and ventricle. Each chamber on one side is


separated from the other by a valve, and it is the closure of


these valves that produce the “lubb-dubb” sound so familiar to


us. (see Fig. 1 – The Structure of the Heart)


Like any other organs in our body, the heart needs a supply


of blood and oxygen, and coronary arteries supply them. There are


two main coronary arteries, the left coronary artery, and the


right coronary artery. They branch off the main artery of the


body, the aorta. The right coronary artery circles the right side


and goes to the back of the heart. The left coronary artery


further divides into the left circumflex and the left anterior


descending artery. These two left arteries feed the front and the


left side of the heart. The division of the left coronary artery


is the reason why doctors usually refer to three main coronary


arteries. (Fig. 2 – Coronary Arteries)


SYMPTOMS OF CORONARY HEART DISEASE


There are three main symptoms of coronary heart disease:


Heart Attack, Sudden Death, and Angina.


Heart Attack


Heart attack occurs when a blood clot suddenly and


completely blocks a diseased coronary artery, resulting in the


death of the heart muscle cells supplied by that artery.


Coronary and Coronary Thrombosis2 are terms that can refer to a


heart attack. Another term, Acute myocardial infarction2, means


death of heart muscle due to an inadequate blood supply.


Sudden Death


Sudden death occurs due to cardiac arrest. Cardiac arrest


may be the first symptom of coronary artery disease and may occur


without any symptoms or warning signs. Other causes of sudden


deaths include drowning, suffocation, electrocution, drug


overdose, trauma (such as automobile accidents), and stroke.


Drowning, suffocation, and drug overdose usually cause


respiratory arrest which in turn cause cardiac arrest. Trauma may


cause sudden death by severe injury to the heart or brain, or by


severe blood loss. Stroke causes damage to the brain which can


cause respiratory arrest and/or cardiac arrest.


Angina


People with coronary artery disease, whether or not they


have had a heart attack, may experience intermittent chest pain,


pressure, or discomforts. This situation is known as angina


pectoris. It occurs when the narrowing of the coronary arteries


temporarily prevents an adequate supply of blood and oxygen to


meet the demands of working heart muscles.


ANGINA PECTORIS


Angina Pectoris (from angina meaning strangling, and


pectoris meaning breast) is commonly known simply as angina and


means pain in the chest. The term “angina” was first used during


a lecture in 1768 by Dr. William Heberden. The word was not


intended to indicate “pain,” but rather “strangling,” with a


secondary sensation of fear.


Victims suffering from angina may experience pressure,


discomfort, or a squeezing sensation in the centre of the chest


behind the breastbone. The pain may radiate to the arms, the


neck, even the upper back, and the pain may come and go. It


occurs when the heart is not receiving enough oxygen to meet an


increased demand.


Angina, as mentioned before, is only temporarily, and it


does not cause any permanent damage to the heart muscle. The


underlying coronary heart disease, however, continues to progress


unless actions are taken to prevent it from becoming worse.


Signs and Symptoms


Angina does not necessarily involve pain. The feeling varies


from individuals. In fact, some people described it as “chest


pressure,” “chest distress,” “heaviness,” “burning feeling,”


“constriction,” “tightness,” and many more. A person with angina


may feel discomforts that fit one or several of the following


descriptions:


- Mild, vague discomfort in the centre of the chest, which


may radiate to the left shoulder or arm


- Dull ache, pins and needles, heaviness or pains in the


arms, usually more severe in the left arm


- Pain that feels like severe indigestion


- Heaviness, tightness, fullness, dull ache, intense


pressure, a burning, vice-like, constriction, squeezing


sensation in the chest, throat or upper abdomen


- Extreme tiredness, exhaustion of a feeling of collapse


- Shortness of breath, choking sensation


- A sense of foreboding or impending death accompanying


chest discomfort


- Pains in the jaw, gums, teeth, throat or ear lobe


- Pains in the back or between the shoulder blades


Angina can be so severe that a person may feel frightened,


or so mild that it might be ignored. Angina attacks are usually


short, from one or two minutes to a maximum of about four to


five. It usually goes away with rest, within a couple of minutes,


or ten minutes at the most.


Different Forms of Angina


There are several known forms of angina. Brief pain that


comes on exertion and leave fairly quickly on rest is known as


stable angina. When angina pain occurs during rest, it is called


unstable angina. The symptoms are usually severe and the coronary


arteries are badly narrowed. If a person suffers from unstable


angina, there is a higher risk for that person to develop heart


attacks. The pain may come up to 20 times a day, and it can wake


a person up, especially after a disturbing dream.


Another type of angina is called atypical or variant angina.


In this type of angina, pain occurs only when a person is resting


or asleep rather than from exertion. It is thought to be the


result of coronary artery spasm, a sort of cramp that narrows the


arteries.


Causes of Angina


The main cause of angina is the narrowing of the coronary


arteries. In a normal person, the inner walls of the coronary


arteries are smooth and elastic, allowing them to constrict and


expand. This flexibility permits varying amounts of oxygenated


blood, appropriate to the demand at the time, to flow through the


coronary arteries. As a person grows older, fatty deposits will


accumulate on the artery walls, especially if the linings of the


arteries are damaged due to cigarette smoking or high blood


pressure.


As more and more fatty materials build up, they form plaques


which causes the arteries to narrow and thus restricting the flow


of blood. This process is known as atherosclerosis. However,


angina usually does not occur until about two-thirds of the


artery’s diameter is blocked. Besides atherosclerosis, there are


other heart conditions resulting in the starvation of oxygen of


the heart, which also causes angina.


The nerve factor – The arteries are supplied with nerves,


which allow them to be controlled directly by the brain,


especially the hypothalamus – an area at the centre of the brain


which regulates the emotions. The brain controls the expanding


and narrowing of the arteries when necessary. The pressures of


modern life: aggression, hostility, never-ending deadlines,


remorseless, competition, unrest, insecurity and so on, can


trigger this control mechanism.


When you become emotional, the chemicals that are released,


such as adrenaline, noradrenaline, and serotonin, can cause a


further constriction of the coronary arteries. The pituitary


gland, a small gland at the base of the brain, under the control


of the hypothalamus, can signal the adrenal glands to increase


the production of stress hormones such as cortisol and adrenaline


even further.


Coronary spasm – Sudden constrictions of the muscle layer in


an artery can cause platelets to stick together, temporarily


restricting the flow of flow. This is known as coronary spasm.


Platelets are minute particles in the blood, which play an


essential role both in the clotting process and in repairing any


damaged arterial walls. They tend to clump together more easily


when the blood is full of chemicals released during arousal, such


as cortisol and others.


Coronary spasm causes the platelets to stick together and to


the wall of the artery, while substances released by the


platelets as they stick together further constrict the blood


vessels. If the artery is already narrowed, this can have a


devastating effect as it drastically reduces the blood flow.


(Fig. 3 – Spasm in a coronary artery)


When people are very tense, they usually overbreathe or hold


their breath altogether. Shallow, irregular but rapid breathing


washes out carbon dioxide from the system and the blood will become


over-oxygenated. One might think that the more oxygen in the blood


the better, but overloaded blood actually does not give up oxygen


as easily, therefore the amount of oxygen available to the heart is


reduced. Carbon dioxide is present in the blood in the form of


carbonic acid, when there is a loss in carbonic acid, the blood


becomes more basic, or alkaline, which leads to spasm of blood


vessels, almost certainly in the brain but also in the heart.


ATHEROSCLEROSIS


The coronary arteries may be clogged with atherosclerotic


plaques, thus narrowing the diameter. Plaques are usually


collections of connection tissue, fats, and smooth muscle cells.


The plaque project into the lumen, the passageway of the artery,


and interfere with the flow of blood. In a normal artery, the


smooth muscle cells are in the middle layer of the arterial wall;


in atherosclerosis they migrate into the inner layer. The reason


behind their migration could hold the answers to explain the


existence of atherosclerosis. Two theories have been developed for


the cause of atherosclerosis.


The first theory was suggested by German pathologist Rudolf


Virchow over 100 years ago. He proposed that the passage of fatty


material into the arterial wall is the initial cause of


atherosclerosis. The fatty material, especially cholesterol, acts


as an irritant, and the arterial wall respond with an outpouring of


cells, creating atherosclerotic plaque.


The second theory was developed by Austrian pathologist Karl


von Rokitansky in 1852. He suggested that atherosclerotic plaques


are aftereffects of blood-clot organization (thrombosis). The clot


adheres to the intima and is gradually converted to a mass of


tissue, which evolves into a plaque.


There are evidences to support the latter theory. It has been


found that platelets and fibrin (a protein, the final product in


thrombosis) are often found in atherosclerotic plaques, also found


are cholesterol crystals and cells which are rich in lipid. The


evidence suggests that thrombosis may play a role in


atherosclerosis, and in the development of the more complicated


atherosclerotic plaque. Though thrombosis may be important in


initiating the plaque, an elevated blood lipid level may accelerate


arterial narrowing.


Plaque


Inside the plaque is a yellow, porridge-like substance,


consisting of blood lipids, cholesterol and triglycerides. These


lipids are found in the bloodstream, they combine with specific


proteins to form lipoproteins. All lipoprotein particles contain


cholesterol, triglycerides, phospholipids, and proteins, but the


proportion varies in different particles.


Lipoproteins


Lipoproteins all vary in size. The largest lipoproteins are


called Chylomicra, and consist mostly of triglycerides. The next in


size are the pre-beta-lipoproteins, then the beta lipoproteins. As


their size decreases, so do their concentration of triglycerides,


but the smaller they are, the more cholesterol they contain. Pre-


beta-lipoproteins are also known as low density lipoproteins (LDL),


and beta lipoproteins are also called very low density lipoproteins


(VLDL). They are most significant in the development of atheroma.


The smallest lipoprotein particles, the alpha lipoproteins, contain


a low concentration of cholesterol and triglycerides, but a high


level of proteins, and are also known as high density lipoproteins


(HDL). They are thought to be protective against the development of


atherosclerotic plaque. In fact, they are transported to the liver


rather than to the blood vessels.


Lipoproteins and Atheroma


The theory is that lipoproteins pass between the lining cells


of the arteries and some of them accumulate underneath. All except


the chylomicra, which are too big, have a chance to accumulate. The


protein in the lipoproteins are broken down by enzymes, leaving


behind the cholesterol and triglycerides. These fats are trapped


and set up a small inflammatory reaction. The alpha particles do


not react with the enzymes are returned to the circulation.


RISK FACTORS


There are several risk factors that contribute to the


development of atherosclerosis and angina: Family history,


Diabetes, Hypertension, Cholesterol, and Smoking.


Family History


We all carry approximately 50 genes that affect the function


and structure of the heart and blood vessels. Genetics can


determine one’s risk of having heart disease. There are many cases


today where heart disease runs in a family, for many generations.


Diabetes


Diabetics are at least twice as likely to develop angina than


nondiabetics, and the risk is higher in women than in men. Diabetes


causes metabolic injury to the lining of arteries, as a result, the


tiny blood vessels that nourish the walls of medium-size arteries


throughout the body, including the coronary arteries, become


defective. These microscopic vessels become blocked, impeding the


delivery of blood to the lining of the larger arteries, causing


them to deteriorate, and artherosclerosis results.


Hypertension


High blood pressure directly injures the artery lining by


several mechanisms. The increased pressure compresses the tiny


vessels that feed the artery wall, causing structural changes in


these tiny arteries. Microscopic fracture lines then develop in the


arterial wall. The cells lining the arteries are compressed and


injured, and can no longer act as an adequate barrier to


cholesterol and other substances collecting in the inner walls of


the blood vessels.


Cholesterol


Cholesterol has become one of the most important issues in the


last decade. Reducing cholesterol intake can directly decrease


one’s risk of developing heart disease, and people today are more


conscious of what they eat, and how much cholesterol their foods


contain.


Cholesterol causes atherosclerosis by progressively narrowing


the arteries and reduces blood flow. The building up of fatty


deposits actually begins at an early age, and the process


progresses slowly. By the time the person reaches middle-age, a


high cholesterol level can be expected.


Smoking


It has been proven that about the only thing smoking do is


shorten a person’s life. Despite all the warnings by the surgeon


general, people still manage to find an excuse to quit smoking.


Cigarette smoke contains carbon monoxide, radioactive


polonium, nicotine, arsenious oxide, benzopyrene, and levels of


radon and molybdenum that are TWENTY times the allowable limit for


ambient factory air. The two agents that have the most significant


effect on the cardiovascular system are carbon monoxide and


nicotine.


Nicotine has no direct effect on the heart or the blood


vessels, but it stimulates the nerves on these structures to cause


the secretion of adrenaline. The increase of adrenaline and


noradrenaline increases blood pressure and heart rate by about 10%


for an hour per cigarette. In simpler words, nicotine causes the


heart to beat more vigorously. Carbon monoxide, on the other hand,


poisons the normal transport systems of cell membranes lining the


coronary arteries. This protective lining breaks down, exposing the


undersurface to the ravages of the passing blood, with all its


clotting factors as well as cholesterol.

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Multiple Risk Factors


The five major risk factors described above do more than just


add to one another. There is a virtual multiplication effect in


victims with more than one risk factor. (Chart: Risk Factors)


DIAGNOSIS


It is very important for patients to tell their doctors of the


symptoms as honestly and accurately as possible. The doctor will


need to know about other symptoms that may distinguish angina from


other conditions, such as esophagitis, pleurisy, costochondritis,


pericarditis, a broken rib, a pinched nerve, a ruptured aorta, a


lung tumour, gallstones, ulcers, pancreatitis, a collapsed lung or


just be nervous. Each of the above mentioned is capable of causing


chest pain.


A patient may take a physical examination, which includes


taking the pulse and blood pressure, listening to the heart and


lung with a stethoscope, and checking weight. Usually an


experienced cardiologist can distinguish it as a cardiac or


noncardiac situation within minutes.


There are also routine tests, such as urine and blood tests,


which can be used to determine body fat level. Blood test can also


tests for:


Anemia – where the level of haemogoblin is too low, and can


restrict the supply of blood to the heart.


Kidney function – levels of various salts, and waste products,


mainly urea and creatinine in the blood. Normally these levels


should be quite low.


There are other factors which can be tested such as salt


level, blood fat and sugar levels.


A chest x-ray provides the doctor with information about the


size of the heart. Like any other muscles in the body, if the heart


works too hard for a period of time, it develops, or enlarges.


An electrocardiogram (ECG) is the tracing of the electrical


activity of the heart. As the heart beats and relaxes, the signals


of the heart’s electrical activities are picked up and the pattern


is recorded. The pattern consists of a series of alternating


plateaus and sharp peaks. ECG can indicate if high blood pressure


has produced any strain on the heart. It can tell if the heart is


beating regularly or irregularly, fast or slow. It can also pick up


unnoticed heart attacks. A variation of the ECG is the


veterocardiogram (VCG). It performs exactly like the ECG except the


electrical activity is shown in the form of loops, or vectors,


which can be watched on a screen, printed on paper, or


photographed. What makes VCG superior to ECG is that VCG provides


a three-dimensional view of a single heart beat.


DRUG TREATMENT


Angina patients are usually prescribed at least one drug. Some


of the drugs prescribed improve blood flow, while others reduce the


strain on the heart. Commonly prescribed drugs are nitrates, beta-


blockers, and Calcium antagonists. It should be noted that drugs


for angina only relief the pain, it does nothing to correct the


underlying disorder.


Nitrates


Nitroglycerine, which is the basis of dynamite, relaxes the


smooth fibres of the blood vessels, allowing the arteries to


dilate. They have a tendency to produce flushing and headaches


because the arteries in the head and other parts of the body will


also dilate.


Glyceryl trinitrate is a short-acting drug in the form of


small tablets. It is taken under the tongue for maximum and rapid


absorption since that area is lined with capillaries. It usually


relieves the pain within a minute or two. One of the drawbacks of


trinitrates is that they can be exposed too long as they


deteriorate in sunlight. Trinitrates also come in the form of


ointment or “transdermal” sticky patch which can be applied to the


skin.


Dinitrates and mononitrates are used for the prevention of


angina attacks rather than as pain relievers. They are slower


acting than trinitrates, but they have a more prolonged effect.


They have to be taken regularly, usually three to four times a day.


Dinitrates are more common than trinitrates or tetranitrates.


Beta-blockers


Beta-blockers are used to prevent angina attacks. They reduce


the work of the heart by regulating the heart beat, as well as


blood pressure; the amount of oxygen required is thereby reduced.


These drugs can block the effects of the stress hormones adrenaline


and noradrenaline at sites called beta receptors in the heart and


blood vessels. These hormones increase both blood pressure and


heart rate. Other sites affected by these hormones are known as


alpha receptors.


There are side effects, however, for using beta-blockers.


Further reduction in the pumping action may drive to a heart


failure if the heart is strained by heart disease. Hands and feet


get cold due to the constriction of peripheral vessels. Beta-


blockers can sometimes pass into the brain fluids, and causes vivid


dreams, sleep disturbance, and depression. There is also a


possibility of developing skin rashes and dry eyes. Some beta-


blockers raise the level of blood cholesterol and triglycerides.


Calcium antagonists


These drugs help prevent angina by moping up calcium in the


artery walls. The arteries then become relaxed and dilated, so


reducing the resistance to blood flow, and the heart receives more


blood and oxygen. They also help the heart muscle to use the oxygen


and nutrients in the blood more efficiently. In larger dose they


also help lower the blood pressure. The drawback for calcium


antagonists is that they tend to cause dizziness and fluid


retention, resulting in swollen ankles.


Other Medications


There are new drugs being developed constantly. Pexid, for


example, is useful if other drugs fail in severe angina attacks.


However, it produces more side effects than others, such as pins


and needles and numbness in limbs, muscle weakness, and liver


damage. It may also precipitate diabetes, and damages to the


retina.


SURGERY


When medications or any other means of treatment are unable to


control the pain of angina attacks, surgery is considered. There


are two types of surgical operation available: Coronary Bypass and


Angioplasty. The bypass surgery is the more common, while


angioplasty is relatively new and is also a minor operation.


Surgery is only a “last resort” to provide relief and should not be


viewed as a permanent cure for the underlying disease, which can


only be controlled by changing one’s lifestyle.


Coronary Bypass Surgery


The bypass surgery involves extracting a vein from another


part of the body, usually the leg, and uses it to construct a


detour around the diseased coronary artery. This procedure restores


the blood flow to the heart muscle.


Although this may sound risky, the death rate is actually


below 3 per cent. This risk is higher, however, if the disease is


widespread and if the heart muscle is already weakened. If the


grafted artery becomes blocked, a heart attack may occur after the


operation.


The number of bypasses depends on the number of coronary


arteries affected. Coronary artery disease may affect one, two, or


all three arteries. If more than one artery is affected, then


several grafts will have to be carried out during the operation.


About 20 per cent of the patients considered for surgery have only


one diseased vessel. In 50 per cent of the patients, there are two


affected arteries, and in 30 per cent the disease strikes all three


arteries. These patients are known to be suffering from triple


vessel disease and require a triple-bypass. Triple vessel disease


and disease of the left main coronary artery before it divides into


two branches are the most serious conditions.


The operation itself incorporates making an incision down the


length of the breastbone in order to expose the heart. The patient


is connected to a heart-lung machine, which takes over the function


of the heart and lungs during the operation and also keeps the


patient alive. At the same time, a small incision is made on the


leg to remove a section of the vein.


Once the section of vein has been removed, it is attached to


the heart. One end of the vein is sewn to the aorta, while the


other end is sewn into the affected coronary artery just beyond the


diseased segment. The grafted vein now becomes the new artery


through which the blood can flow freely beyond the obstruction. The


original artery is thus bypassed. The whole operation requires


about four to five hours, and may be longer if there is more than


one bypass involved. After the operation, the patient is sent to


the Intensive Care Unit (ICU) for recovery.


The angina pain is usually relieved or controlled, partially


or completely, by the operation. However, the operation does not


cure the underlying disease, so the effects may begin to diminish


after a while, which may be anywhere from a few months to several


years. The only way patients can avoid this from happening is to


change their lifestyles.


Angioplasty


This operation is a relatively new procedure, and it is known


in full as transluminal balloon coronary angioplasty. It entails


“squashing” the atherosclerotic plaque with balloons. A very thin


balloon catheter is inserted into the artery in the arm or the leg


of a patient under general anaesthetic. The balloon catheter is


guided under x-ray just beyond the narrowed coronary artery. Once


there, the balloon is inflated with fluid and the fatty deposits


are squashed against the artery walls. The balloon is then deflated


and drawn out of the body.


This technique is a much simpler and more economical


alternative to the bypass surgery. The procedure itself requires


less time and the patient only remains in the hospital for a few


days afterward. Exactly how long the operation takes depends on


where and in how many places the artery is narrowed. It is most


suitable when the disease is limited to the left anterior


descending artery, but sometimes the plaques are simply too hard,


making them impossible to be squashed, in which case a bypass might


be necessary.


SELF-HELP


The only way patients can prevent the condition of their heart


from deteriorating any further is to change their lifestyles.


Although drugs and surgery exist, if the heart is exposed to


pressure continuously and it strains any further, there will come


one day when nothing works, and all that remain is a one-way ticket


to heaven.


The following are some advices on how people can change the


way they live, and enjoy a lifetime with a healthy heart once more.


Work


A person should limit the amount of exertions to the point


where angina might occur. This varies from person to person, some


people can do just as much work as they did before developing


angina, but only at a slower pace. Try to delegate more, reassess


your priorities, and learn to pace yourself. If the rate of work is


uncontrollable, think about changing the job.


Exercise


Everyone should exercise regularly to one’s limits. This may


sound contradictory that, on the one hand, you are told to limit


your exertion and, on the other, you are told to exercise. It is


actually better if one exercise regularly within his or her limits.


Exercises can be grouped into two categories: isotonic and


isometric. People suffering from angina should limit themselves to


only isotonic exercises. This means one group of muscle is relaxed


while another group is contracted. Examples of this type of


exercise include walking, swimming leisurely, and yoga; some harder


exercises are cycling and jogging.


Weight Loss


The more weight there is on the body, the more work the heart


has to do. Reducing unnecessary weight will reduce the amount of


strain on the heart, and likely lower blood pressure as well. One


can lose weight by simply eating less than their normal intake, but


keep in mind that the major goal is to cut down on fatty and sugar


foods, which are low in nutrients and high in calories.


Diet


What you eat can have a direct effect on the kind of condition


you are in. To stay fit and healthy, eat fewer animal fats, and


foods that are high in cholesterol. They include fatty meat, lard,


suet, butter, cream and hard cheese, eggs, prawns, offal and so on.


Also, the amount of salt intake should be reduced. Eat more food


containing a high amount of fibre, such as wholegrain cereal


products, pulses, wholemeal bread, as well as fresh fruits and


vegetables.


Alcohol, tea and coffee


Alcohol in moderation does no harm to the body, but it does


contain calories and may slow the weight loss progress. People can


drink as much mineral water, fruit juice and ordinary or herb tea


as they wish, but no more than two cups of coffee per day.


Cigarettes


It has been medically proven that cigarettes do the body no


good at all. It makes the heart beat faster, constricts the blood


vessels, and generally increases the amount of work the heart has


to do. The only right thing to do is to quit smoking, it will not


be easy, but it is worth the effort.


Stress


Stress can actually be classified as a major risk factor, and


it is one neglected by most people. Try to avoid those heated


arguments and emotional situations that increase blood pressure, as


well as stimulate the release of stress hormones. If they are


unavoidable, try to anticipate them and prevent the attack by


sucking an angina tablet beforehand.


Relaxation


Help your body to relax when feeling tense by sitting or lying


down quietly. Close your eyes, breathe slowly and deeply through


the nose, make each exhalation long, soft and steady. An adequate


amount of sleep each night is always important.


Sexual activity


It is true that sexual intercourse may bring on an angina


attack, but the chronic frustration of abstinence may cause more


tension. If intercourse precipitates angina, either suck on an


angina tablet a few minutes beforehand or let your partner assume


the more active role.


TYPE-A BEHAVIOUR PATTERN


There is a marked increase of coronary heart disease in most


industrialized societies in the twentieth century. This may have


resulted, in part, because these societies reward those who


performed more quickly, aggressively, and competitively.


Type-A individuals of both sexes were considered to have the


following characteristics:


(1) an intense, sustained drive to achieve self-


selected but often poorly defined goals.


(2) a profound inclination and eagerness to compete.


(3) a persistent desire for recognition and


advancement.


(4) a continuous involvement in multiple and diverse


functions subject to time restrictions.


(5) habitual propensity to accelerate the rate of


execution of most physical and mental functions.


(6) extraordinary mental and physical alertness.


(7) aggressive and hostile feelings.


The enhanced competitiveness of type-A persons leads to an


aggressive and ambitious achievement orientation, increased mental


and physical alertness, muscular tension, and an explosive and


rapid style of speech. A sense of time urgency leads to


restlessness, impatience, and acceleration of most activities. This


in turn may result in irritability and the enhanced potential for


type-A hostility and anger. Type-A individuals are thus at an


increased risk of developing coronary heart disease.


The type-A behaviour pattern is defined as an action-emotion


complex involving10:


(1) behavioural dispositions (e.g., ambitiousness,


aggressiveness, competitiveness, and impatience).


(2) specific behaviours (e.g., muscle tenseness,


alertness, rapid and emphatic speech stylistics,


and accelerated pace of most activities).


(3) emotional responses (e.g., irritation, hostility,


and anger).


Comparatively, type-A persons are more risky to develop


coronary heart disease than type-B individuals, whose manners and


behaviours are relaxed. The risk, however, is independent of the


risk factors. Not all physicians are convinced that type-A


behaviour pattern is a risk factor, and thousands of studies and


researches are currently being done by experts on this topic.


THE CARDIAC REHAB PROGRAM


This program at the Credit Valley Hospital is designed to help


patients with coronary artery disease lower their overall risk, and


to prevent any further attacks. It provides rehabilitation for


patients who are likely to have heart attacks, have had heart


attacks, or had a recent surgery.


Most patients come to this one-hour class two nights a week,


which takes place outside the physiotherapy department. The class


is ran by volunteers, and is usually supervised by a kinesiologist.


The patients come in a little before 6:00 pm, and have their blood


pressure taken. At six o’clock, volunteers will take the patients


through a fifteen-minute warm-up. After the warm-up, the patients


will go on with their exercise for half an hour. The patients can


choose from walking, rowing machines, stationary bicycles, and arm


ergometer, or a combination of two or more as their exercise.


Each patient is reassessed once a month, in order to keep


track of their progress. Volunteers will ask the patient being


reassessed a series of questions, which includes frequency of


exercise, type of exercise program, problems with exercise, etc.


About 6:30, when the patients are near the peak of their


exercise, the ones being reassessed will have to have their pulse


and blood pressure measured; to see if they have reached their


“target heart rate”, and to see if their blood pressure goes up


as expected.


At about 6:45, the patients end their exercise and cool-down


begins. Cool-down is in a way similar to warm-up, only this helps


the patients to relax their hearts, as well as their body after a


half-hour workout. After cool-down most patients have their blood


pressure taken again just to make sure nothing unusual occurs.


CONCLUSION


Angina pectoris is not a disease which affect a person’s


heart permanently, but to encounter angina pain means something


is wrong. The pain is the heart’s distress signal, a built-in


warning device indicating that the heart has reached its maximum


workload. Upon experiencing angina, precautions should be taken.


A person’s lifestyle plays a major role in determining the


chance of developing heart diseases. If people do not learn how


to prevent it themselves, coronary artery disease will remain as


the single biggest killer in the world, by far.

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