РефератыИностранный языкAiAids Essay Research Paper AidsAcquired Immune Deficiency

Aids Essay Research Paper AidsAcquired Immune Deficiency

Aids Essay, Research Paper


Aids


Acquired Immune Deficiency Syndrome (AIDS), suppresses the immune


system related to infection with the human immunodeficiency virus (HIV). A person


infected with HIV gradually loses immune function along with certain immune cells


called CD4 T-lymphocytes or CD4 T-cells, causing the infected person to become


vulnerable to pneumonia, fungus infections, and other common ailments. With the loss


of immune function, a clinical syndrome (a group of various illnesses that together


characterize a disease) develops over time and eventually results in death due to


opportunistic infections (infections by organisms that do not normally cause disease


except in people whose immune systems have been greatly weakened) or cancers.


In the early 1980s deaths by opportunistic infections, previously observed


mainly in organ transplant recipients receiving therapy to suppress their immune


responses, were recognized in otherwise healthy homosexual men. In 1983, French


cancer specialist Luc Montagnier and scientists at the Pasteur Institute in Paris isolated


what appeared to be a new human retrovirus?a special type of virus that reproduces


differently from other viruses?from the lymph node of a man at risk for AIDS. Nearly


simultaneously, scientists working in the laboratory of


American research scientist Robert Gallo at the National Cancer Institute in Bethesda,


Maryland, and a group headed by American virologist Jay Levy at the University of


California at San Francisco isolated a retrovirus from people with AIDS and


individuals having contact with people with AIDS. All three groups of scientists


isolated what is now known as human immunodeficiency virus (HIV), the virus that


causes AIDS.


Infection with HIV does not necessarily mean that a person has AIDS, although


people who are HIV-positive are often mistakenly said to have AIDS. In fact, a person


can remain HIV-positive for more than ten years without developing any of the clinical


illnesses that define and constitute a diagnosis of AIDS. In 1996 an estimated 22.6


million people worldwide were living with HIV or AIDS?21.8 million adults and 830,000 children. The World Health Organization (WHO) estimates that between 1981, when the first AIDS cases were reported, and the end of 1996, more than 8.4 million adults and children had developed AIDS. In this same period there were 6.4 million deaths worldwide from AIDS or HIV. About 360,000 of these deaths occurred in the United States.


Clinical Progression of AIDS


The progression from the point of HIV infection to the clinical diseases that


define AIDS may take six to ten years or more. This progression can be monitored


using surrogate markers (laboratory data that correspond to the various stages of


disease progression) or clinical endpoints (illnesses associated with more advanced


disease). Surrogate markers for the various stages of HIV infection include the


declining number of CD4 T-cells, (the major type of white blood cell lost because of


HIV infection). In general, the lower the infected person?s CD4 T-cell count, the


weaker the person?s immune system and the more advanced the disease state. In 1996, it


became evident that the actual amount of HIV in a person?s blood?the so-called viral


burden?could be used to predict the progression to Aids, regardless of a person?s


CD4 T-cell count. With advancing technology, Viral Burden Determinations are


quickly becoming a standard means of patient testing. An infected person?s immune


response to the virus?that is, the person?s ability to produce antibodies against HIV?


can also be used to determine the progression ofAids; however, this surrogate marker is less precise during more advanced stages of


AIDS because of the overall loss of immune function.


Within one to three weeks after infection with HIV, most people experience


nonspecific flulike symptoms such as fever, headache, skin rash, tender lymph nodes,


and a vague feeling of discomfort. These symptoms last about one to two weeks.


During this phase, known as the acute retroviral syndrome phase, HIV reproduces to


very high concentrations in the blood, mutates (changes its genetic nature) frequently,


circulates through the blood, and establishes infections throughout the body, especially


in the lymphoid organs. The infected person?s CD4 T-cell count falls briefly but then


returns to near normal levels as the person?s immune system responds to the infection.


Individuals are thought to be highly infectious during this phase. <

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Following the acute retroviral syndrome phase, infected individuals enter a


prolonged asymptomatic phase?a symptom-free phase that can last ten years or more.


Persons with HIV remain in good health during this period, with levels of CD4 T-cells


ranging from low to normal (500 to 750 cells per cubic mm of blood). Nevertheless,


HIV continues to replicate during the asymptomatic phase, causing progressive


destruction of the immune system. Eventually, the immune system weakens to the point


that the person enters the early symptomatic phase. This phase can last from a few


months to several years and is characterized by rapidly falling levels of CD4 T-cells


(500 to 200 cells per cubic mm of blood) and opportunistic infections that are not life


threatening. Following the early symptomatic phase, the infected person experiences the


extensive immune destruction and serious illness that characterize the late symptomatic


phase. This phase can also last from a few months to years, and the affected individual


may have CD4 T-cell levels below 200 per cubic mm of blood along with certain


opportunistic infections that define AIDS. A wasting syndrome of progressive weight


loss and debilitating fatigue occurs in a large proportion of people in this stage. The


immune system is in a state of severe failure. The person eventually enters the


advanced AIDS phase, in which CD4 T-cell numbers are below 50 per cubic mm of


blood. Death due to severe life-threatening opportunistic infections and cancers usually


occurs within one to two years.


Opportunistic Illnesses


Death from AIDS is generally due not to HIV infection itself, but to


opportunistic infections that occur when the immune system can no longer protect the


body against agents normally found in the environment. The appearance of any one of


more than 25 different opportunistic infections, called AIDS-defining illnesses, along


with a CD4 T-cell count of less than 200 cells per cubic millimeter of blood provides


the clinical diagnosis of AIDS in HIV-infected individuals.


The most common opportunistic infection seen in AIDS is Pneumocystis Carinii


Pneumonia (PCP), which is caused by a fungus that normally exists in the airways of all


people. Bacterial Pneumonia and Tuberculosis are also commonly associated with


AIDS. In the late symptomatic phase of AIDS, bacterial infection by Mycobacterium


avium can cause fever, weight loss, anemia, and diarrhea. Additional bacterial


infections of the gastrointestinal tract commonly cause diarrhea, weight loss, anorexia


(loss of appetite), and fever. Also, during advanced AIDS, diseases caused by protozoal


parasites, especially Toxoplasmosis of the nervous system, are common.


In addition to PCP, people with AIDS often develop other fungal infections.


Thrush, an infection of the mouth by the fungus Candida Albicans, is common in the


early symptomatic phase of AIDS. Other infectious fungi include species of the genus


Cryptococcus, a major cause of Meningitis in up to 13 percent of people with AIDS.


Also, infection by the fungus Histoplasma Capsulatum affects up to 10 percent of


people with AIDS, causing general weight loss, fever, and respiratory complications or


severe central nervous system complications if the infection reaches the brain.


Viral opportunistic infections, especially with members of the Herpes virus


family, are common in people with AIDS. One Herpes family member, Cytomegalovirus


(CMV), infects the retina of the eye and can result in blindness. Another herpes virus,


Epstein-Barr virus (EBV), may result in a cancerous transformation of blood cells.


Infections with Herpes Simplex Virus types 1 and 2 are also common and result


in progressive sores around the mouth and anus.


Many people with AIDS develop cancers, the most common types being B-cell


Lymphoma and Kaposi?s Sarcoma. Kaposi?s Sarcoma?a cancer of blood vessels


that results in purple lesions on the skin that can spread to internal organs and cause


death?occurs mainly in homosexual and bisexual men. Although the cause of KS is


unknown, a link between KS and a new type of herpes virus was discovered in 1994.


Human Immunodeficiency Virus (HIV)


The causative agent of AIDS is HIV, a human retrovirus. Researchers have


known since 1984 that HIV enters human cells by binding with a receptor protein known


as CD4, located on human immune-cell surfaces. HIV carries on its surface a viral


protein known as cp120, which specifically recognizes and binds

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